beyond reason
0교시 체육수업 네이퍼빌 학생 - 운동화 신은 뇌(클릭클릭)
운동을 하고 공부를 하면 성적이 오른다
운동은 신경가소성을 증진시켜 치매를 예방, 치료한다
One potential mediator of exercise effects is the neurotrophin BDNF, which enhances neuroplasticity via different pathways (e.g., synaptogenesis, neurogenesis, long-term potentiation).
- 운동은 신경유래성장인자를 유도하여 시냅스 형성, 신경재생, 장기강화 등의 pathway를 자극하고 신경가소성을 일으킴.
운동을 하면
(i) increased peripheral lactate levels (following high intensity exercise) are associated with increased peripheral BDNF levels,
(ii) lactate infusion at rest can increase peripheral and central BDNF levels and
(iii) lactate plays a very complex role in the brain’s metabolism.
- 젖산수치를 높여 BDNF수치를 높임
- 젖산주입은 bdnf수치를 높임
- 젖산은 뇌의 대사에 복합역할을 함.
file:///Users/anandamoon/Downloads/jcm-09-01136-v3.pdf
Review
Lactate and BDNF: Key Mediators of Exercise Induced Neuroplasticity?
Abstract:
Accumulating evidence from animal and human studies supports the notion that physical exercise can enhance neuroplasticity and thus reduce the risk of several neurodegenerative diseases (e.g., dementia). However, the underlying neurobiological mechanisms of exercise induced neuroplasticity are still largely unknown. One potential mediator of exercise effects is the neurotrophin BDNF, which enhances neuroplasticity via different pathways (e.g., synaptogenesis, neurogenesis, long-term potentiation).
Current research has shown that
(i) increased peripheral lactate levels (following high intensity exercise) are associated with increased peripheral BDNF levels,
(ii) lactate infusion at rest can increase peripheral and central BDNF levels and
(iii) lactate plays a very complex role in the brain’s metabolism.
In this review, we summarize the role and relationship of lactate and BDNF in exercise induced neuroplasticity.